Melatonin induces cell apoptosis in Mia PaCa-2 cells via the suppression of nuclear factor-κB and activation of ERK and JNK: A novel therapeutic implication for pancreatic cancer
AddexBio Product Detail - MIA PaCa-2 Cells
PLOS ONE: MicroRNA Profiling Implies New Markers of Gemcitabine Chemoresistance in Mutant p53 Pancreatic Ductal Adenocarcinoma
Cancer Cell Lines
CX-5461 induces senescence in A375 and MIA Paca-2 cancer cells. A375... | Download Scientific Diagram
Spheroid cells in MIA PaCa-2, Panc-1, and PC-3. A, Pancreatic cancer... | Download Scientific Diagram
Upregulation of the splice variant MUC4/Y in the pancreatic cancer cell line MIA PaCa-2 potentiates proliferation and suppresses apoptosis: New insight into the presence of the transcript variant of MUC4
AKT inhibition is associated with chemosensitisation in the pancreatic cancer cell line MIA-PaCa-2 | British Journal of Cancer
JCRB0070:MIA PaCa-2
Melatonin induces cell apoptosis in Mia PaCa-2 cells via the suppression of nuclear factor-κB and activation of ERK and JNK: A novel therapeutic implication for pancreatic cancer
Mia-PaCa-2
JCRB0070:MIA PaCa-2
The Cell Line-Dependent Diversity in Initial Morphological Dynamics of Pancreatic Cancer Cell Peritoneal Metastasis Visualized by an Artificial Human Peritoneal Model - ScienceDirect
JCRB0070:MIA PaCa-2
Human Cell Lines-Cell Lines-InCellGene LLC.-CellLines,Biomedicine reasearch Product,Cell Biology,Cell Culture,293T Cell,Media
A repetitive mutation and selection system for bacterial evolution to increase the specific affinity to pancreatic cancer cells | bioRxiv
Portal do Conhecimento do Departamento de Bioquímica
ces-03-267-g006
MIA PaCa-2 and PANC-1 – pancreas ductal adenocarcinoma cell lines with neuroendocrine differentiation and somatostatin receptors | Scientific Reports
Gatifloxacin causes activation of p21 in MIA PaCa-2 and p27 in Panc-1 cells, but Skp2 in both cell lines.
Targeting the Janus-activated kinase-2-STAT3 signalling pathway in pancreatic cancer using the HSP90 inhibitor ganetespib - ScienceDirect
